Question

哪位好心人帮我翻译下英语翻译日语，谢谢啦！要写论文用！

Low-grade inflammation has also been shown to play a role in the development of the pathogenesis associated with obesity .lt has been suggested that lipopolysaccharide (LPS) Was involved in the development of the metabolic syndrome associated with high-fat feeding . These reports described that mice fed on a high-fat diet for 2 to 4 weeks exhibited a significant increase in plasma LPS. This increase in LPS in the plasma induced by a high-fat diet suggests metabolic endotoxemia, Which is considered to trigger the development of obesity , inflammation , insulin resistance , type 2 diabetes and atherosclerosis via activation of the CD14/TLR4 receptor complex by LPS and/or the fatty acids. Furthermore , it has also been demonstrated that the population levels of Bifidobacterium spp . and the Eubacterium rectal /clostridium coccoides group were significantly reduced in high-fat-fed mice as compared to those receiving the standard high carbohydrate diet .The administration of oligofructose (OFS) to high-fat-fed mice may thus abolish the increase in plasma LPS With associated decreases in energy intake , body Weight gain ,glycemia and epididymal fat mass. The Bifidobacterium spp.levela were significantly increased in OFS-administered mice , while the plasma LPS levels decreased.
All these findings strongly suggest that gut microbiota contribute to the complications related to high-fat diet feeding . it would thus be useful to develop specific strategies using prebiotics for modifying gut microbiota to favor specific gut microbiota and to prevent the delirious effects of high-fat or obesity-induced complications. Bifidobacteria are gram-positive anaerobic microorganisms that naturally colonize the human intestinal tract and are believed to be beneficial to human health . Several previous studies have shown that bifidobacteria lowered the intestinal endotoxin level and improved the mucosal barrier function . However, the effect ot administering specific probiotics added to high – fat diet-induced obesity has not been defined . we evaluated in the present study the effect of an oral administration of the bifidobacterial strain ,Bifidobacterium breve B-3, in a mouse model with high-fat diet –induced obesity and insulin resistance.

Answer 1

軽度炎症もは证明されていただく役」と発展の発病のメカニズムは、肥満と関系があります。ltは脂多糖(lps)が、代谢症侯群の発展に関わった脂肪の多い食べ物を与えず。これらの报告、高脂肪の食べ物をネズミに饵を2 ~ 4周间に、著しい増加プラズマ有限を得た。このような成长がプラズマによる有限事业者が高脂肪食建议代谢エンドトキシン、こちらは肥満?メタボリックの発展、炎症、インスリン抵抗、2型糖尿病やアテローム性动脉硬化活力回复を通じて受容体cd14 / tlr4の复雑な有限业者と/あるいは脂肪酸だ。また、人口の水准であることを明らかにしたビフィズス菌をふやし菌sppだった。和真菌便意/ソロ状バジラスcoccoides组の减少が顕著と高脂肪に饵ネズミに比べ、受け入れ基准の高炭水化物食べ物だ。政府の低聚果糖(ofs)高脂肪に饵ラットだろうというのが大方の廃止と减少と関连した血浆にlpsエネルギー摂取し、体は体重を増やし、血糖値と附睾の脂肪量だ。ビフィズス菌をふやし菌spp . levelaシチューとofsはるかに向上させることで管理し、プラズマlps含有量が低くなった。
このすべての発见强くの可能性を示唆した肠微生物群による合并症で高脂肪食。そのためには特定の策略の开発を使用益生元改正肠微生物群に特定の肠微生物群と防止精神错乱の影响を受けた高脂肪か肥満诱导合并症だ。ビフィズス菌をふやし菌はグラム阳性の「嫌酸素微生物、自然と人间膓とは「人体の健康に良い。一部の先の研究をすでに明らかにし、ビフィズス菌をふやし菌を下げる肠エンドトキシン水准と改善した粘膜の障壁が机能である。しかし、効果を管理できない特定の益生菌添加t

Answer 2

建议楼主去找专业人士翻译，太长了。翻译这个要花很多精力，别人一点开就吓死了。再有就是把这些分开来多问几个问题，短了可能还有人愿意帮你翻译。